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Cancer-associated fibroblasts’ EXOs lncRNA NEAT1 promotes the growth of endometrial cancer through the STAT3/YKL-40 signaling pathway controlled by miR-26a/b-5p (Fan et al., 2021)

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More broadly, the present findings establish SMG1 as a multifunctional stress-response kinase whose inhibition simultaneously disrupts NRF2-dependent redox buffering, NMD-mediated suppression of tumor neoantigen expression, and Regnase-1-dependent inflammatory mRNA decay to remodel the tumor microenvironment—three mechanistically independent outputs converging on a single molecular target

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